myopericarditis guidelines
Email © American Heart Association, Inc. All rights reserved. This site uses cookies. After a physical examination, your doctor might order one or more tests to confirm that you have myocarditis and determine its severity. 2015 ESC Guidelines for the diagnosis and management of pericardial diseases. However, randomized clinical trials of antiviral therapy have not yet established viruses as therapeutic targets in acute myocarditis. The need for a worldwide registry. ESC Clinical Practice Guidelines aim to present all the relevant evidence to help physicians weigh the benefits and risks of a particular diagnostic or therapeutic procedure on Pericardial Diseases. Myocarditis treatment focuses on the cause and the symptoms, such as heart failure.In mild cases, persons should avoid competitive sports for at least three to six months. the myocarditis treatment trial investigators.Left ventricular assist device support and myocardial recovery in recent onset cardiomyopathy.2015 SCAI/ACC/HFSA/sts clinical expert consensus statement on the use of percutaneous mechanical circulatory support devices in cardiovascular care: Endorsed by the American Heart Assocation, the Cardiological Society of India, and Sociedad Latino Americana de Cardiologia Intervencion; Affirmation of Value by the Canadian Association of Interventional Cardiology-Association Canadienne de Cardiologie d’intervention.Extracorporeal membranous oxygenation support for acute fulminant myocarditis: analysis of a single center’s experience.Extracorporeal membrane oxygenation for the support of adults with acute myocarditis.Venoarterial extracorporeal membrane oxygenation for acute fulminant myocarditis in adult patients: a 5-year multi-institutional experience.Short-term mechanical circulatory support as a bridge to durable left ventricular assist device implantation in refractory cardiogenic shock: a systematic review and meta-analysis.Favourable clinical outcome in patients with cardiogenic shock due to fulminant myocarditis supported by percutaneous extracorporeal membrane oxygenation.Mechanical unloading by fulminant myocarditis: LV-IMPELLA, ECMELLA, BI-PELLA and PROPELLA concepts[published online August 6, 2018].Integrins and integrin-related proteins in cardiac fibrosis.Cardiac fibroblasts support cardiac inflammation in heart failure.Inflammation - cause or consequence of heart failure or both?Facilitated cardiac recovery in fulminant myocarditis: pediatric use of the Impella LP 5.0 pump.Successful use of the Impella device in giant cell myocarditis as a bridge to permanent left ventricular mechanical support.Fulminant myocarditis managed by extracorporeal life support (Impella® CP): a rare case.Novel combination of impella and extra corporeal membrane oxygenation as a bridge to full recovery in fulminant myocarditis.Combined use of Impella left ventricular assist device and extracorporeal membrane oxygenation as a bridge to recovery in fulminant myocarditis.Impella and extracorporeal membrane oxygenation: a demanding combination.Concomitant implantation of Impella® on top of veno-arterial extracorporeal membrane oxygenation may improve survival of patients with cardiogenic shock.Full percutaneous biventricular support with two Impella pumps: the Bi-Pella approach.Mode-of-action of the PROPELLA concept in fulminant myocarditis.Hemodynamic effects of immunoadsorption and subsequent immunoglobulin substitution in dilated cardiomyopathy: three-month results from a randomized study.Immunoadsorption in dilated cardiomyopathy: long-term reduction of cardiodepressant antibodies.Long-term benefits of immunoadsorption in β(1)-adrenoceptor autoantibody-positive transplant candidates with dilated cardiomyopathy.TNF-α-secreting B cells contribute to myocardial fibrosis in dilated cardiomyopathy.B-cell activation influences T-cell polarization and outcome of anti-CD20 B-cell depletion in central nervous system autoimmunity.B lymphocytes trigger monocyte mobilization and impair heart function after acute myocardial infarction.Rituximab reversed cardiac involvement of Wegener’s granulomatosis: magnetic resonance imaging assessment.B-cell epitope spreading is a critical step for the switch from C-protein-induced myocarditis to dilated cardiomyopathy.When cancer and heart failure cross paths: a case report of severe cardiorenal compromise associated with the anti-CD20 monoclonal antibody rituximab in a patient with dilated cardiomyopathy.Successful treatment of cardiac involvement in dermatomyositis with rituximab.Lupus myocarditis receiving the rituximab therapy-a monocentric retrospective study.Rituximab in recurrent idiopathic giant cell myocarditis after heart transplantation: a potential therapeutic approach.Rituximab is associated with improved survival in cardiac allograft patients with antibody-mediated rejection: a single center review.Betaferon in chronic viral cardiomyopathy (BICC) trial: Effects of interferon-β treatment in patients with chronic viral cardiomyopathy.Interferon-Beta improves survival in enterovirus-associated cardiomyopathy.Interferon beta modulates endothelial damage in patients with cardiac persistence of human parvovirus b19 infection.Aggravation of left ventricular dysfunction in patients with biopsy-proven cardiac human herpesvirus A and B infection.Human herpesvirus-6: a short review of its biological behavior.Chromosomally integrated human herpesvirus 6 in heart failure: prevalence and treatment.Viral myocarditis: a prime example for endomyocardial biopsy-guided diagnosis and therapy.High prevalence of cardiac parvovirus B19 infection in patients with isolated left ventricular diastolic dysfunction.Parvovirus B19-induced vascular damage in the heart is associated with elevated circulating endothelial microparticles.Human parvovirus B19 NS1 protein modulates inflammatory signaling by activation of STAT3/PIAS3 in human endothelial cells.Impaired endothelial regeneration through human parvovirus b19-infected circulating angiogenic cells in patients with cardiomyopathy.Fatal parvovirus B19 myocarditis in an 8-year-old boy.High prevalence of human parvovirus B19 DNA in myocardial autopsy samples from subjects without myocarditis or dilative cardiomyopathy.Low level myocardial parvovirus B19 persistence is a frequent finding in patients with heart disease but unrelated to ongoing myocardial injury.Bioportfolio: lifelong persistence of variant and prototypic erythrovirus DNA genomes in human tissue.Molecular phenotypes of human parvovirus B19 in patients with myocarditis.Differences in virus prevalence and load in the hearts of patients with idiopathic dilated cardiomyopathy with and without immune-mediated inflammatory diseases.A distinct subgroup of cardiomyopathy patients characterized by transcriptionally active cardiotropic erythrovirus and altered cardiac gene expression.Relevance of cardiac parvovirus B19 in myocarditis and dilated cardiomyopathy: review of the literature.Recapitulation of IVIG anti-inflammatory activity with a recombinant IgG Fc.Intravenous immunoglobulin therapy for patients with idiopathic cardiomyopathy and endomyocardial biopsy-proven high PVB19 viral load.Efficacy of telbivudine in the treatment of chronic hepatitis b and liver cirrhosis and its effect on immunological responses.Telbivudine improves the function of myeloid dendritic cells in patients with chronic hepatitis B.Telbivudine therapy may shape CD4(+) T-cell response to prevent liver fibrosis in patients with chronic hepatitis B.Effect of telbivudine therapy on the cellular immune response in chronic hepatitis B.Telbivudine in chronic lymphocytic myocarditis and human parvovirus B19 transcriptional activity.Prevalence of erythrovirus genotypes in the myocardium of patients with dilated cardiomyopathy.Productive infection by B19 parvovirus of human erythroid bone marrow cells Immunosuppression in lymphocytic myocarditis with parvovirus B19 presence.Effects of interleukin-1 blockade with anakinra on adverse cardiac remodeling and heart failure after acute myocardial infarction [from the Virginia Commonwealth University-Anakinra Remodeling Trial (2) (VCU-ART2) pilot study].Interleukin-1 blockade in acute decompensated heart failure: a randomized, double-blinded, placebo-controlled pilot study.Effects of interleukin-1 blockade with anakinra on aerobic exercise capacity in patients with heart failure and preserved ejection fraction (from the D-HART pilot study).Interleukin-1 receptor blockade rescues myocarditis-associated end-stage heart failure.Treating Life-Threatening Myocarditis by Blocking Interleukin-1.Antiinflammatory therapy with canakinumab for atherosclerotic disease.Colchicine in addition to conventional therapy for acute pericarditis: results of the COlchicine for acute PEricarditis (COPE) trial.Colchicine for recurrent pericarditis (CORP): a randomized trial.A randomized trial of colchicine for acute pericarditis.COlchicine for the Prevention of the Post-pericardiotomy Syndrome (COPPS): a multicentre, randomized, double-blind, placebo-controlled trial.Colchicine for prevention of postpericardiotomy syndrome and postoperative atrial fibrillation: the COPPS-2 randomized clinical trial.Critical role of RAGE and HMGB1 in inflammatory heart disease.Identification of human S100A9 as a novel target for treatment of autoimmune disease via binding to quinoline-3-carboxamides.Pathogenic role of the damage-associated molecular patterns S100A8 and S100A9 in coxsackievirus b3-induced myocarditis.Endomyocardial proteomic signature corresponding to the response of patients with dilated cardiomyopathy to immunoadsorption therapy.Adoptive transfer of regulatory T cells protects against Coxsackievirus B3-induced cardiac fibrosis.Regulatory T cells protect mice against coxsackievirus-induced myocarditis through the transforming growth factor beta-coxsackie-adenovirus receptor pathway.Administration of regulatory T cells ameliorates myocardial inflammation in experimental myocarditis.Immunosuppression with an interleukin-2 fusion protein leads to improved LV function in experimental ischemic cardiomyopathy.Mesenchymal stem cells improve murine acute coxsackievirus B3-induced myocarditis.Mesenchymal stromal cells but not cardiac fibroblasts exert beneficial systemic immunomodulatory effects in experimental myocarditis.Immunomodulatory effects of mesenchymal stromal cells revisited in the context of inflammatory cardiomyopathy.Mesenchymal stromal cells modulate monocytes trafficking in coxsackievirus b3-induced myocarditis.Mesenchymal stromal cells inhibit NLRP3 inflammasome activation in a model of Coxsackievirus B3-induced inflammatory cardiomyopathy.Intramyocardial transplantation of cardiac mesenchymal stem cells reduces myocarditis in a model of chronic Chagas disease cardiomyopathy.Intramyocardial bone marrow mononuclear cells versus bone marrow-derived and adipose mesenchymal cells in a rat model of dilated cardiomyopathy.Randomized comparison of allogeneic versus autologous mesenchymal stem cells for nonischemic dilated cardiomyopathy: POSEIDON-DCM Trial.Human cardiac-derived adherent proliferating cells reduce murine acute Coxsackievirus B3-induced myocarditis.NOD2 (Nucleotide-Binding Oligomerization Domain 2) is a major pathogenic mediator of coxsackievirus b3-induced myocarditis.NLRP3 inflammasome activation: the convergence of multiple signalling pathways on ROS production?Formation of the inflammasome in acute myocarditis.Involvement of NLRP3 inflammasome in CVB3-induced viral myocarditis.The Nlrp3 inflammasome promotes myocardial dysfunction in structural cardiomyopathy through interleukin-1β.Blocking the IL-1β signalling pathway prevents chronic viral myocarditis and cardiac remodeling.Inhibition of interleukin-1 by anakinra improves vascular and left ventricular function in patients with rheumatoid arthritis.Interleukin-1 blockade with anakinra to prevent adverse cardiac remodeling after acute myocardial infarction (Virginia Commonwealth University Anakinra Remodeling Trial [VCU-ART] Pilot study).Interleukin-1 blockade in recently decompensated systolic heart failure: results from REDHART (Recently Decompensated Heart Failure Anakinra Response Trial).IL-1 blockade in patients with heart failure with preserved ejection fraction.Effect of anakinra on recurrent pericarditis among patients with colchicine resistance and corticosteroid dependence: the airtrip randomized clinical trial.Rationale and design of the virginia commonwealth university-anakinra remodeling trial-3 (VCU-ART3): a randomized, placebo-controlled, double-blinded, multicenter study.Gout-associated uric acid crystals activate the NALP3 inflammasome.Colchicine–Update on mechanisms of action and therapeutic uses.2015 ESC Guidelines for the diagnosis and management of pericardial diseases: the task force for the diagnosis and management of pericardial diseases of the European Society of Cardiology (ESC)Endorsed by: The European Association for Cardio-Thoracic Surgery (EACTS).Colchicine reduces NLRP3 inflammasome activity in murine Coxsackievirus B3-induced myocarditis.Microtubule inhibition therapy by colchicine in severe myocarditis especially caused by Epstein-Barr and cytomegalovirus co-infection during a two-year period: a novel therapeutic approach.Colchicine aggravates coxsackievirus B3 infection in mice.The Janus face of HMGB1 in heart disease: a necessary update.An overview on HMGB1 inhibitors as potential therapeutic agents in HMGB1-related pathologies.MRP8 and MRP14 control microtubule reorganization during transendothelial migration of phagocytes.The endogenous Toll-like receptor 4 agonist S100A8/S100A9 (calprotectin) as innate amplifier of infection, autoimmunity, and cancer.Differential release and deposition of S100A8/A9 proteins in inflamed upper airway tissue.Plasma S100A8/A9 correlates with blood neutrophil counts, traditional risk factors, and cardiovascular disease in middle-aged healthy individuals.Elevated serum calprotectin levels in HIV-infected patients: the calprotectin response during ZDV treatment is associated with clinical events.CX3CR1 knockout aggravates Coxsackievirus B3-induced myocarditis.S100A8 and S100A9 induce cytokine expression and regulate the NLRP3 inflammasome via ROS-dependent activation of NF-κB(1.
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